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complete culture medium human oral epithelial cells  (Procell Inc)

 
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    Procell Inc complete culture medium human oral epithelial cells
    WNT7B promotes the migration and invasion of OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B is overexpressed in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis that detected <t>epithelial</t> to mesenchymal transition marker E-cadherin protein levels decreased and the N-cadherin protein increased in HN6 and HN30 cells after WNT7B overexpressed. GAPDH served as a normalization control for WB. n = 3 independent experiments; error bars, mean ± SD; ns, not significant, * P < 0.05, ** P < 0.01, *** P < 0.001; t-test
    Complete Culture Medium Human Oral Epithelial Cells, supplied by Procell Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/complete culture medium human oral epithelial cells/product/Procell Inc
    Average 90 stars, based on 1 article reviews
    complete culture medium human oral epithelial cells - by Bioz Stars, 2026-02
    90/100 stars

    Images

    1) Product Images from "WNT7B promotes cancer progression via WNT/β-catenin signaling pathway and predicts a poor prognosis in oral squamous cell carcinoma"

    Article Title: WNT7B promotes cancer progression via WNT/β-catenin signaling pathway and predicts a poor prognosis in oral squamous cell carcinoma

    Journal: BMC Oral Health

    doi: 10.1186/s12903-024-05113-9

    WNT7B promotes the migration and invasion of OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B is overexpressed in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis that detected epithelial to mesenchymal transition marker E-cadherin protein levels decreased and the N-cadherin protein increased in HN6 and HN30 cells after WNT7B overexpressed. GAPDH served as a normalization control for WB. n = 3 independent experiments; error bars, mean ± SD; ns, not significant, * P < 0.05, ** P < 0.01, *** P < 0.001; t-test
    Figure Legend Snippet: WNT7B promotes the migration and invasion of OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B is overexpressed in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis that detected epithelial to mesenchymal transition marker E-cadherin protein levels decreased and the N-cadherin protein increased in HN6 and HN30 cells after WNT7B overexpressed. GAPDH served as a normalization control for WB. n = 3 independent experiments; error bars, mean ± SD; ns, not significant, * P < 0.05, ** P < 0.01, *** P < 0.001; t-test

    Techniques Used: Migration, Wound Healing Assay, Invasion Assay, Western Blot, Marker, Control

    WNT7B inhibited epithelial to mesenchymal transition in OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B was knockdown in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis detecting epithelial to mesenchymal transition marker E-cadherin protein levels increased and N-cadherin protein levels decreased in HN6/HN30 cells after WNT7B knockdown. GAPDH served as a normalization control for WB. Error bars, mean ± SD; n = 3 independent experiments; error bars, mean ± SD; * P < 0.05, ** P < 0.01, *** P < 0.001; t-test
    Figure Legend Snippet: WNT7B inhibited epithelial to mesenchymal transition in OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B was knockdown in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis detecting epithelial to mesenchymal transition marker E-cadherin protein levels increased and N-cadherin protein levels decreased in HN6/HN30 cells after WNT7B knockdown. GAPDH served as a normalization control for WB. Error bars, mean ± SD; n = 3 independent experiments; error bars, mean ± SD; * P < 0.05, ** P < 0.01, *** P < 0.001; t-test

    Techniques Used: Wound Healing Assay, Migration, Invasion Assay, Knockdown, Western Blot, Marker, Control



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    completed culture medium for human oral epithelial cells  (Procell Inc)
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    PB2 inhibited viability and promoted apoptosis of OSCC cells. (a) The chemical structure of PB2. (b) The viability of oral mucosa <t>epithelial</t> cell (OMEC) treated with PB2 was detected by CCK-8 assay. (c) The viability of human OSCC cell line (SCC-25) treated with PB2 was detected by CCK-8 assay. (d) The apoptosis of SCC-25 cells treated with PB2 was analyzed by Tunel assay. (e) The expression of apoptosis related proteins in SCC-25 cells treated with PB2 was determined by Western blot. *P < 0.05, **P < 0.01 and ***P < 0.001 vs. PB2 (0 μM) group.
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    Image Search Results


    WNT7B promotes the migration and invasion of OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B is overexpressed in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis that detected epithelial to mesenchymal transition marker E-cadherin protein levels decreased and the N-cadherin protein increased in HN6 and HN30 cells after WNT7B overexpressed. GAPDH served as a normalization control for WB. n = 3 independent experiments; error bars, mean ± SD; ns, not significant, * P < 0.05, ** P < 0.01, *** P < 0.001; t-test

    Journal: BMC Oral Health

    Article Title: WNT7B promotes cancer progression via WNT/β-catenin signaling pathway and predicts a poor prognosis in oral squamous cell carcinoma

    doi: 10.1186/s12903-024-05113-9

    Figure Lengend Snippet: WNT7B promotes the migration and invasion of OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B is overexpressed in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis that detected epithelial to mesenchymal transition marker E-cadherin protein levels decreased and the N-cadherin protein increased in HN6 and HN30 cells after WNT7B overexpressed. GAPDH served as a normalization control for WB. n = 3 independent experiments; error bars, mean ± SD; ns, not significant, * P < 0.05, ** P < 0.01, *** P < 0.001; t-test

    Article Snippet: Human oral epithelial cells, H203, were purchased from Procell Life Science & Technology Co., Ltd. (Procell, Wuhan, #CP-H203) and maintained in a complete culture medium for human oral epithelial cells (Procell, Wuhan, #CM-H203).

    Techniques: Migration, Wound Healing Assay, Invasion Assay, Western Blot, Marker, Control

    WNT7B inhibited epithelial to mesenchymal transition in OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B was knockdown in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis detecting epithelial to mesenchymal transition marker E-cadherin protein levels increased and N-cadherin protein levels decreased in HN6/HN30 cells after WNT7B knockdown. GAPDH served as a normalization control for WB. Error bars, mean ± SD; n = 3 independent experiments; error bars, mean ± SD; * P < 0.05, ** P < 0.01, *** P < 0.001; t-test

    Journal: BMC Oral Health

    Article Title: WNT7B promotes cancer progression via WNT/β-catenin signaling pathway and predicts a poor prognosis in oral squamous cell carcinoma

    doi: 10.1186/s12903-024-05113-9

    Figure Lengend Snippet: WNT7B inhibited epithelial to mesenchymal transition in OSCC cells. ( A - D ) Wound-healing assay, the migration and invasion assay analyses of changes in migratory capacity as WNT7B was knockdown in HN6/HN30 cells. Bars, 500 μm. ( E - H ) Western blot analysis detecting epithelial to mesenchymal transition marker E-cadherin protein levels increased and N-cadherin protein levels decreased in HN6/HN30 cells after WNT7B knockdown. GAPDH served as a normalization control for WB. Error bars, mean ± SD; n = 3 independent experiments; error bars, mean ± SD; * P < 0.05, ** P < 0.01, *** P < 0.001; t-test

    Article Snippet: Human oral epithelial cells, H203, were purchased from Procell Life Science & Technology Co., Ltd. (Procell, Wuhan, #CP-H203) and maintained in a complete culture medium for human oral epithelial cells (Procell, Wuhan, #CM-H203).

    Techniques: Wound Healing Assay, Migration, Invasion Assay, Knockdown, Western Blot, Marker, Control

    PB2 inhibited viability and promoted apoptosis of OSCC cells. (a) The chemical structure of PB2. (b) The viability of oral mucosa epithelial cell (OMEC) treated with PB2 was detected by CCK-8 assay. (c) The viability of human OSCC cell line (SCC-25) treated with PB2 was detected by CCK-8 assay. (d) The apoptosis of SCC-25 cells treated with PB2 was analyzed by Tunel assay. (e) The expression of apoptosis related proteins in SCC-25 cells treated with PB2 was determined by Western blot. *P < 0.05, **P < 0.01 and ***P < 0.001 vs. PB2 (0 μM) group.

    Journal: Bioengineered

    Article Title: Procyanidin B2 inhibits angiogenesis and cell growth in oral squamous cell carcinoma cells through the vascular endothelial growth factor (VEGF)/VEGF receptor 2 (VEGFR2) pathway

    doi: 10.1080/21655979.2022.2033013

    Figure Lengend Snippet: PB2 inhibited viability and promoted apoptosis of OSCC cells. (a) The chemical structure of PB2. (b) The viability of oral mucosa epithelial cell (OMEC) treated with PB2 was detected by CCK-8 assay. (c) The viability of human OSCC cell line (SCC-25) treated with PB2 was detected by CCK-8 assay. (d) The apoptosis of SCC-25 cells treated with PB2 was analyzed by Tunel assay. (e) The expression of apoptosis related proteins in SCC-25 cells treated with PB2 was determined by Western blot. *P < 0.05, **P < 0.01 and ***P < 0.001 vs. PB2 (0 μM) group.

    Article Snippet: OMEC was cultured in the completed culture medium for human oral epithelial cells (Procell).

    Techniques: CCK-8 Assay, TUNEL Assay, Expressing, Western Blot

    PB2 inhibited invasion, migration and epithelial-mesenchymal transition (EMT) of OSCC cells. The migration (a) and invasion (b) of SCC-25 cells treated with PB2 were detected by wound healing assay and transwell assay. The expression of metastasis associated proteins (c) and EMT related proteins (d) was analyzed by Western blot. *P < 0.05, **P < 0.01 and ***P < 0.001 vs. PB2 (0 μM) group.

    Journal: Bioengineered

    Article Title: Procyanidin B2 inhibits angiogenesis and cell growth in oral squamous cell carcinoma cells through the vascular endothelial growth factor (VEGF)/VEGF receptor 2 (VEGFR2) pathway

    doi: 10.1080/21655979.2022.2033013

    Figure Lengend Snippet: PB2 inhibited invasion, migration and epithelial-mesenchymal transition (EMT) of OSCC cells. The migration (a) and invasion (b) of SCC-25 cells treated with PB2 were detected by wound healing assay and transwell assay. The expression of metastasis associated proteins (c) and EMT related proteins (d) was analyzed by Western blot. *P < 0.05, **P < 0.01 and ***P < 0.001 vs. PB2 (0 μM) group.

    Article Snippet: OMEC was cultured in the completed culture medium for human oral epithelial cells (Procell).

    Techniques: Migration, Wound Healing Assay, Transwell Assay, Expressing, Western Blot